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chip-it express enzymatic shearing kit  (Active Motif)


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    Structured Review

    Active Motif chip-it express enzymatic shearing kit
    Chip It Express Enzymatic Shearing Kit, supplied by Active Motif, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/chip-it%C2%AE+express+shearing+kit/chip+it+express+enzymatic+shearing+kit/pmc12129438-65-5-15
    Average 90 stars, based on 1 article reviews
    chip-it express enzymatic shearing kit - by Bioz Stars, 2026-11
    90/100 stars

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    Related Articles

    Chromatin Immunoprecipitation:

    Article Title: The regulatory G4 motif of the Kirsten ras (KRAS) gene is sensitive to guanine oxidation: implications on transcription.
    Article Snippet: Chromatin immunoprecipitation (ChIP) and qPCR analysis ChIP was carried out as described in ref. 31, by using the ChIP-IT® Express Shearing Kit (Active Motif, CA, USA).

    Article Title: The ROS-KRAS-Nrf2 axis in the control of the redox homeostasis and the intersection with survival-apoptosis pathways: Implications for photodynamic therapy.
    Article Snippet: In highly proliferating cancer cells oncogenic mutations reprogram the metabolism and increase the production of reactive oxygen species (ROS).. Cancer cells prevent ROS accumulation by upregulating antioxidant systems.. Here we show that an increase of oxidative stress (ROS and singlet oxygen), generated by photoactivated TMPyP4, results in the upregulation of KRAS and Nrf2, the major regulator of the redox homeostasis.

    Real-time Polymerase Chain Reaction:

    Article Title: The regulatory G4 motif of the Kirsten ras (KRAS) gene is sensitive to guanine oxidation: implications on transcription.
    Article Snippet: Chromatin immunoprecipitation (ChIP) and qPCR analysis ChIP was carried out as described in ref. 31, by using the ChIP-IT® Express Shearing Kit (Active Motif, CA, USA).

    Article Title: The ROS-KRAS-Nrf2 axis in the control of the redox homeostasis and the intersection with survival-apoptosis pathways: Implications for photodynamic therapy.
    Article Snippet: In highly proliferating cancer cells oncogenic mutations reprogram the metabolism and increase the production of reactive oxygen species (ROS).. Cancer cells prevent ROS accumulation by upregulating antioxidant systems.. Here we show that an increase of oxidative stress (ROS and singlet oxygen), generated by photoactivated TMPyP4, results in the upregulation of KRAS and Nrf2, the major regulator of the redox homeostasis.



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